Gene: KCNK3 - part of the brain that it impacts is the arucate nucleus 1. what is the function of this gene? In other words, What is the protein product and what does this protein do? 2. How might a mutation in this gene alter the development of the arucate nucleus in a way that alters gender identity?

Gene: KCNK3 - part of the brain that it impacts is the arucate nucleus 1. what is the function of this gene? In other words, What is the protein product and what does this protein do? 2. How might a mutation in this gene alter the development of the arucate nucleus in a way that alters gender identity?

Biology: The Dynamic Science (MindTap Course List)

4th Edition

ISBN:9781305389892

Author:Peter J. Russell, Paul E. Hertz, Beverly McMillan

Publisher:Peter J. Russell, Paul E. Hertz, Beverly McMillan

Chapter56: Animal Behavior

Section: Chapter Questions

Problem 15TYK

See similar textbooks

Similar questions

Discuss Concepts In the 1930s and 1940s, prefrontal lobotomy, in which neural connections in the frontal lobes of both cerebral hemispheres were severed, was used to treat behavioral conditions such as extreme anxiety and rebellious- ness. Although the procedure calmed patients, it had side effects such as apathy and a seriously disrupted personality. In view of the information presented in this chapter, why do you think the operation had these effects?
Which of the following is NOT true about the specification of the eye field? (Check all that apply) A ET induces RX, which in turn induces Otx2 and Pax6. B During late gastrulation, the forebrain and hindbrain are specified. Pax6 initiates the expression of a network of transcription factors, some of which mutually activate one another. D ET induces RX, which in turn induces Pax6 and blocks Otx2 within the eye field. ET induces RX, which in turn blocks Otx2 and Pax6. F During early gastrulation, Noggin promotes the expression of both ET and Otx2. During late gastrulation, Otx2 blocks the inhibition of ET by Noggin. H) It starts with neural induction when the neural plate is induced within the ectoderm. Pax6 is only required for the specification of the retina. During early gastrulation, Noggin indices Otx2 but blocks ET. E Feedback That's not correct
| What role does the synaptonemal complex play? How is this complex different from cohesins?
This is one given problem on my homework assignment for developmental biology. Thank you for the help!Describe the effects of FGF and RA on each other in limb bud development. Give a specific example of their effects on each other in this capacity. Knowing this, propose a question/hypothesis to investigate the mechanism of how a specific morphogen controls pattern if you know that the morphogen is both necessary and sufficient AND is expressed in the correct spatial/temporal frame.
How would mutations that completely ablate the function of the androgen receptor impact the phenotypic development of humans with XY chromosomes? Patients would appear phenotypically female Patients would appear phenotypically male with underdeveloped secondary sex characteristics Patients would appear phenotypically male, but cannot produce sperm Patients would express both male and female secondary sex characteristics.
Animal Models: The Search for Behavior Genes You are a researcher studying an autosomal dominant neurodegenerative disorder. You have cloned the gene underlying the disorder and have found that it encodes an enzyme that is overexpressed in the neurons of individuals who have the disorder. To better understand how this enzyme causes neurodegeneration in humans, you make a strain of transgenic Drosophila whose nerve cells overexpress the enzyme. a. How might you use these transgenic flies to try to gain insight into the disease or identify drugs that might be useful in the treatment of the disease? b. Can you think of any potential limitations of this approach?
5. If an experimenter were to express light-activated channelrhodopsin in a mouse using the Bhlhb5 promoter, what behavioral outcome would support the current model for responses to itchy vs. painful stimuli? nociceptive stimull skin DRG nociceptive TRPV1+ pruriceptive TRPV11/MrprA3+ spinal cord GRP GRP Bhib5 GRPR pain Itch
19: What are 3 phenotypes of GA, or BR, or ABA, or CK over-producing mutants. 20: Think about a hormone receptor, specifically the structure of the receptor. Describe ONE why a mutation would cause insensitivity to the hormone, AND ONE way it could cause hypersensitivity to the hormone. Explain your answer to each. 22: Not all cells in tissues are the same. They differentiate from stem cells. Describe how ONE cell type acquires cell identity. (Pick any cell in the plant)
7. Fill in the table below, outlining different aspects of glutamate singlaing in the mammalian brain. A legend of possible entries is provided below the table, specifically for the columns "marker genes", “associated G proteins" and "ionotropic targets" Marker gene Ionotropic receptors Possible associated G proteins Gs Gą G₁/Go Possible marker genes: Choline acetyltransferase Solute carrier family 17 Glutamate decarboxylase Dopamine ß hydroxylase Possible ionotropic targets: Cayl channels Cav2 channels GIRK channels M-type K¹ channels BK channels SK channels Metabotropic Associated receptors G protein mGluR 1,5 mGluR 2-4,6-7- Pre- or post- Ionotropic Neuronal effects synaptic action? target(s) Tryptophan hydroxylase 2 Tyrosine hydroxylase Histidine decarboxylase
In rats, the spinal nucleus of the bulbocavernosus undergoes greater a) apoptosis; females b) apoptosis; males c) neurogenesis; males d) neurogenesis; females in
9. We have the following DNA sequence that codes for 7 amino acids of exon 1 of the dopamine receptor gene. 5’- GTG CAC CTG ACT CCT GAG AAG -3’ a. Obtain the sequence of the complementary strand to said DNA sequence: b. Obtain the sequence of the messenger RNA and indicate which direction it leads: c. Using the genetic code obtain the protein sequence: d). What effect would it have on the cells, in terms of their receptor for dopamine, if there was a stop codon in the third position in that mRNA sequence?
A Na+ 10 msec B 40mV Ca²+ C Na+ Ca²+ Each graph represents the depolarization activity during an action potential in Xenopus spinal cells at different stages of development. Referring to each electrical reading (A, B, and C), place the graphs in order of developmental stage, from earliest to latest. What are the major changes in action potential activity that occurs with development?