3. Figure on the right shows a DNAGenes expressedmicroarrayassayofgenein cancerousexpressionlevels.Inthistissue: Redmicroarray if a sample withnormal tissue is labeled withGenes expressedin normal tissue:green fluorescent dye, and aGreensample with cancerous tissue islabeled with red, what coloredGenes expressedin both tissues:spots would represent genes thatyou would be interested in if youYellowwere studying cancer? Explain.Genes expressedin neither tissue:Black

Micro arrays are utilised for the determination of gene expression patterns in specific tissues or…

3. Figure on the right shows a DNA microarray assay of gene expression levels. In this microarray if a sample with normal tissue is labeled with green fluorescent dye, and a sample with cancerous tissue is labeled with red, what colored spots would represent genes that you would be interested in if you were studying cancer? Explain.

3. Figure on the right shows a DNA microarray assay of gene expression levels. In this microarray if a sample with normal tissue is labeled with green fluorescent dye, and a sample with cancerous tissue is labeled with red, what colored spots would represent genes that you would be interested in if you were studying cancer? Explain.

Biology 2e

2nd Edition

ISBN:9781947172517

Author:Matthew Douglas, Jung Choi, Mary Ann Clark

Publisher:Matthew Douglas, Jung Choi, Mary Ann Clark

Chapter17: Biotechnology And Genomics

Section: Chapter Questions

Problem 3VCQ: Figure 17.15 In 2011, the United States Preventative Services Task Force recommended against using...

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Our government has finite funds to devote to cancer research.Discuss which of the following areas of research you think shouldreceive the most funding.A. Identifying and characterizing oncogenes and tumorsuppressorgenesB. Identifying agents in our environment that cause cancerC. Identifying viruses that cause cancer D. Devising methods aimed at killing cancer cells in the bodyE. Informing the public of the risks involved in exposure tocarcinogensIn the long run, in which of these areas would you expect successfulresearch to be the most effective in decreasing human mortalitydue to cancer?
E DBOW GENERAL-BIOLOGY-1.ec x A genbio1_mod2_-Cell-Types and x O File | C/Users/Acer/Desktop/genbio1/genbio1_mod2_-Cell-Types%20and%20Cell%20Modifications%20(1).pdf ... D Page view | A Read aloud V Draw E Highlight 12 of 20 Erase What are nervous tissues? How does the nervous tissue work? What makes up a typical nerve cell or neuron? Dendrite Cell body Axon Nervous tissue senses stimuli and transmits signals called nerve impulses from one part of an animal to another. It has a cell body, containing the cell's nucleus, and several slender extensions. One type of extension, called a dendrite, conveys signals from its tip toward the rest of the neuron; another type, the axon, transmits signals toward another neuron or to a muscle cell. What's More Helow is a picture that showa the organs of a human torso. Identify the tissue that ia found in each labeled part of the torso. Inger HUure: VeciurSuk File name: Human anatomy model with different aystems Vector Image 9 10:32 am P Type here to…
Proto-oncogenes can be converted to oncogenes in a numberof different ways. In some cases, the proto-oncogene itselfbecomes amplified up to hundreds of times in a cancer cell.An example is the cyclin D1 gene, which is amplified in somecancers. In other cases, the proto-oncogene may be mutatedin a limited number of specific ways, leading to alterations inthe gene product’s structure. The ras gene is an example of aproto-oncogene that becomes oncogenic after suffering pointmutations in specific regions of the gene. Explain why thesetwo proto-oncogenes (cyclin D1 and ras) undergo such differentalterations to convert them into oncogenes
1. Match each of the terms in the left column to the bestfitting phrase from the right column.a. epistatic interaction 1. divide the body into identical units(segments)b. regulative 2. initiated by binding of ligand todetermination receptorc. modifier screen 3. individuals with cells of more thanone genotyped. RNAi 4. the fate of early embryonic cells canbe altered by the environmente. ectopic expression 5. assign identity to body segmentsf. homeodomain 6. substance whose concentrationdetermines cell fatesg. green fluorescent 7. suppression of gene expression byprotein double-stranded RNAh. genetic mosaics 8. method for identifying pleiotropicgenesi. segmentation genes 9. a DNA-binding motif found incertain transcription factorsj. homeotic genes 10. encode proteins that accumulate inunfertilized eggs and are needed forembryo developmentk. morphogen 11. double mutant has phenotype of oneof the two mutantsl. maternal effect 12. a gene is turned on in an inappropriategenes tissue or at…
of my celle chose are wels smaller than normal. According to Model 1, what part(e) of the cell cycle is (ere) ou likely being affected? Some cancerus me 7. In Model 1, if the length of the arrow represents time, then for those cancerous cells, what hap pens to the time that is necessary for the cell cycle? What implication might this have for doctors who are treating cancer patients? Why? Living things must grow and develop. At times they suffer injuries or damage, or cells simply wear out. New cells must be formed for the organism to survive. What process must occur to make a new, properly- functioning cell? Model 1 - Mitosis as Part of the Cell Cycle Prophase Metaphase Anaphase Mitosis How do living things grow and repair themselves! Replicated chromosome (2 sister chromatids) 0000 * Centriole Spindle fibers Nuclear membrane Telophase 1. Refer to Model 1. List the four phases in the mitosis process. 2. Where is mitosis in the cell cycle? Before 3. What three phases of the cell cycle…
Many human cancers result when a normal gene mutates and leads to uncontrolled growth (a tumor). Genesthat cause cancer when they mutate are called oncogenes. Chemotherapy is effective against many tumorsbecause it targets rapidly dividing cells and kills them.Unfortunately, chemotherapy has many side effects,such as hair loss or nausea, because it also kills many ofour normal cells that are rapidly dividing, such as thosein the hair follicles or stomach lining.Many scientists and large pharmaceutical companiesare excited about the prospects of exploiting theRNAi pathway to selectively inhibit oncogenes in lifethreatening tumors. Explain in very general terms howgene-silencing therapy might work to treat cancer andwhy this type of therapy would have fewer side effectsthan chemotherapy
You have treated undifferentiated embryonic stem cells (ESCs) with sonic hedgehog (Shh)for several days. This should induce a neural fate in your ESCs and you hope that you havebeen successful in differentiating the ESCs into NSCs. What is one assay you could use to testwhether your cells had differentiated into NSC-like cells? Name and shortly describe the assay?
A. Describe the location of the following: Describe the location 1Chromosome 2.Deoxynibonucieic Acid (ONA) 3Gene B. Label the different parts of the cell. 2. waten by OONCIOHN M HERRER C jute juno puede" Umido, Junto aanza con el EduKalidad
. Mutations in an autosomal gene in humans cause aform of hemophilia called von Willebrand disease(vWD). This gene specifies a blood plasma proteincleverly called von Willebrand factor (vWF). vWFstabilizes factor VIII, a blood plasma protein specified by the wild-type hemophilia A gene. Factor VIIIis needed to form blood clots. Thus, factor VIII is rapidly destroyed in the absence of vWF.Which of the following might successfully be employed in the treatment of bleeding episodes in hemophiliac patients? Would the treatments workimmediately or only after some delay needed forprotein synthesis? Would the treatments have only ashort-term or a prolonged effect? Assume that allmutations are null (that is, the mutations result in thecomplete absence of the protein encoded by the gene)and that the plasma is cell-free.a. transfusion of plasma from normal blood into avWD patientb. transfusion of plasma from a vWD patient into adifferent vWD patientc. transfusion of plasma from a hemophilia A…
1. A brief intrwhich introduces ALP as a diagnostic tool in cancer and create a Graphical Abstract to summarise the role of ALP in cancer?
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// Which CDK and cyclin does p53 ultimately target in DNA damaged conditions? O CDK1/cyclin E O CDK2/cyclin E CDK1/cyclin B CDK2/cyclin B