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- The enzyme is considered to be alan * ÇOO ÇOO Lactate dehydrogenase HO-C-H+ NAD C=0 + NADH + H CH: Pyruvate Lactate O Oxydoreductase O Isomerase O Hydrolase O Ligase O DehydrataseDiscuss the metabolic control for HMG CoA reductase synthesis and activityint IMLeagues | Provi... Time Remaining: 00:18:21 A Hide Time Remaining A Arsenic is a water soluble element that is a poison because it functions as an allosteric inhibitor of the citric acid cycle enzymes pyruvate dehydrogenase and succinate dehydrogenase. One of the consequences of arsenic poisoning is lactic acidosis from the buildup of lactate in the body. Why does arsenic treatment result in a buildup of lactate? Legend Pyruvate Acetyl Hydrogen COA SH + NAD+ Pyruvate dehydrogenase Co,+NADH, H* Adenosine Carbon ATR triphosphate CoA Охygen Guanosine CGTP triphosphate Sulfur Acetyl-CoA COA SH Q Coenzyme Q CoA Coenzyme A HCO3 +CATP Water NADH Nicotinamide adenine dinucleotide Citrate Pyruvate carboxylase Pyruvate dehydrogenase Enzyme ADP+Pi Citrate synthase Aconitase Oxaloacetate Water cis-Aconitate Water NADH, H* Aconitase Malate dehydrogenase NAD. D-Isocitrate NAD* Malate NADH, H* Isocitrate dehydrogenase CO2 Citric acid cycle Fumarase MacBook Pr
- The oxidation of Gly-3-P to 1,3-bisphosphoglycerate proceeds with an unfavorable AG which equals 6.3 KJ/mol, yet the flow through this point in the glycolytic pathway proceeds smoothly. Why???Handwritten Identify the molecule names, enzyme name, enzyme classification and change in reaction(for glycolysis pathway)HANDOUT C Glucose-6 Phosphate Dehydrogenase and TPI Deficiencies Glucose-6-P GSH + GSH Reduced glutathione 2 H+ NADP+ H₂O₂ Hydrogen peroxide Glucose-6-phosphate dehydrogenase Superoxide radical NADPH + H+ 6-Phosphogluconolactone GSSG Glutathione Oxidized reductase Glutathione peroxidase Aerobic respiration, drugs, fava beans glutathione 2 H₂O Detoxification SOD: superoxide dismutase GPx: glutathione peroxidase HK: hexokinase GSSG and GSH: glutathione, oxidized and reduced forms, respectively CAT: catalase The diagram above gives important details about the HMP shunt. Superoxides/free radicals dis- rupt membrane lipids, causing changes in membrane permeability. The HMP shunt is expressed in liver cells, red blood cells, and in lipid-producing tissues. 1. Discuss how glycolysis is affected in G6PD patients, using the diagrams to explain your response. 2. How does this effect differ from TPI patients? 3. Why are red blood cells, but not the nervous system, affected in G6PD patients?