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how did the initial reaction to florence nightingate and semmelweis contributed to the resistance to improve iniatives?
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- How was thermal stability measured in this paper and describe how GRL-1720 and 5h affect the thermal stability of Mpro and which drug has the larger effect? https://www.nature.com/articles/s41467-021-20900-6What is infusion related toxicity and slower toxicity of amphotericin B? How can these be minimized?You perform Michaelis-Menten kenetics on i) trypsin and ii) a mutant form of the same enzyme (a single amino acid has been changed). The specificity constant for the mutant was 10 times larger than for trypsin. a) Define the specificity constant and explain using this definition how a larger value might occur. b) Explain how, if at all, a non-competitive inhibitor of trypsin would affect the specificity constant.
- What is neomycin phosphotransferase (designated neor)?Serum blood of a patient with dislipoproteinemia type 1 has milky appearance even in fasting. If serum stays at low temperature (40) for several hours fatty layer appears on its surface. What are the possible causes of these symptoms? To explain this, answer the questions and do the following tasks: a) what compounds of serum must be tested for that patient in biochemical lab? b) write the reaction which does not occur properly in patient’s blood; c) write down the schemes, explaining how the products of the previous reaction are used in adipose tissue and heart in healthy person 2 hours after a meal.What exactly are prodrugs? How do enzyme inhibition and induction impact the therapeutic action of a prodrug?
- What are the limitations of Etelcalcetide, and how can the structure of the drug be modified to address these limitations?What is the biochemical basis of allopurinol treatment for gout?Under what conditions, a higher rate in the presence of the inhibitor observed? (how could the effect of inhibitor be compensated) explain
