What are the observed genotypic frequencies for Wildrose at the pt-8 locus? and What are the observed allelic frequencies for Wildrose at the pt-8 locus?
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Genetic Variation
Genetic variation refers to the variation in the genome sequences between individual organisms of a species. Individual differences or population differences can both be referred to as genetic variations. It is primarily caused by mutation, but other factors such as genetic drift and sexual reproduction also play a major role.
Quantitative Genetics
Quantitative genetics is the part of genetics that deals with the continuous trait, where the expression of various genes influences the phenotypes. Thus genes are expressed together to produce a trait with continuous variability. This is unlike the classical traits or qualitative traits, where each trait is controlled by the expression of a single or very few genes to produce a discontinuous variation.
I'm confused about how I would figure out these two questions as I don't really understand the difference or where to start. What are the observed genotypic frequencies for Wildrose at the pt-8 locus? and What are the observed allelic frequencies for Wildrose at the pt-8 locus?
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- 2. 235O 4) Q7. Haemophiliacs possess a non-functional form of the gene responsible for the production of blood clotting factors. Shown below is the occurrence of haemophilia in one family. = male = female = male haemophiliac 7. 8. 5. 9. 3. 11 12 Usingthe following symbols: H = dominant allele h = recessive allele 1) State the genotypes of the following individuals. Individual Genotype 1. 6. 2) On the basis of the information provided, is the inheritance of haemophilia: (i) autosomal or sex-linked? (ii) dominant or recessive? 3) State the probability of individual 8 being a carrier of haemophilia. 4) Explain why only females can be carriers of haemophilia.W O () ENG 9:37 am O GENBIO-1ST-SEM-MIDTERN X 9 Schoology G karyotype of a certain huma x 6 BigBlueButton - GNBIO Messenger My Questions | bartleby + A app.schoology.com/common-assessment-delivery/start/5385424680?action=Donresume&submissionld=643190401 The diagram below shows a karyotype of a certain human. 8. 10 11 12 13 14 15 16 17 18 19 21 22 X Y Based on the karyotype, which of the following statements is most likely true? O The individual has a genetic condition caused by a nondisjunction event. The individual has a genetic condition caused by the X and Y chromosomes being different sizes. O The individual has a genetic condition caused by a chromosomal duplication. O The individual has a genetic condition caused chromosomes number one being different sizes. GENBIO-1ST-SEM-.pdf O 245180335_56899...jpg Show all ... TID N DAD × IDD . I ID.9/assessments/424097/variants/623602/take/6/ dity Test 4 Points SKETCHPAD Question 7 Use the Punnett square in the sketch pad below to cross two heterozygous tall plants to determine the possible offspring. Let R tall and r = short. Rr Rr C«» 0:00/0:12 Tall X Tall (Mating) Speed: 1x A Q Paused
- Corn id W20-AB1-D-13 486 152 162 54 W21-BB1-A-19 445 152 154 94 W20-BB1-D-19 501 153 167 88 W20-AB1-C-16 489 153 94 56 W20-BB1-H-11 480 153 94 56 MONOHYBRID CROSS1. Present the data as monohybrids (Xx, XX, xx) for all five corn cobs andfor the plant. Present the colour and texture phenotypes with their associatedgenotypes in separate tables. Your first table should include the corn ID, the number of purple kernels, thenumber of yellow kernels, and the phenotypic ratio between the two kernelcolours. Present the texture results in the same format to a separate table.2. What is the dominant phenotype for kernel colour in your assigned cornplant? What is the dominant phenotype for kernel texture in your assigned corn plant?3. What are the probable phenotypes for the previous generation of cornplants with regard to kernel coloration?4. What are the probable phenotypes of the previous generation of cornplants with regard to kernel texture?Bruce Ames and his colleagues have pointed out that although detailed toxicological analysis has been conducted on synthetic chemicals, almost no information is available about the mutagenic or carcinogenic effects of the toxins produced by plants as a natural defense against fungi, insects, and animal predators. Tens of thousands of such compounds have been discovered, and he estimates that in the United States adults eat about 1.5 g of these compounds each daylevels that are approximately 10,000 times higher than those of the synthetic pesticides present in the diet. For example, cabbage contains 49 natural pesticides and metabolites, and only a few of these have been tested for their carcinogenic and mutagenic effects. a. With the introduction of new foods into the U.S. diet over the last 200 years (mangoes, kiwi fruit, tomatoes, and so forth), has there been enough time for humans to develop resistance to the mutagenic effects of the toxins present in those foods? b. The natural pesticides present in plants constitute more than 99% of the toxins we eat. Should diet planning, especially for vegetarians, take into account the doses of toxins present in the diet?- Gramm X P Pearson X G translat x 7 Traduct x Textboc x b Bongo x b Virt X Unit 58 3144/viewContent/112835/View pruce Harbor, Mai.. Aprende a hablar u. speaking, how many genes are involved in influencing such characters? b. How do the measurements of quantitative characters distribute in a population, i.e., according to what kind of curve? c. Give an example of a 2 quantitative characters other than those mentionedin the class materials. 1) 2) Q4. A person has symptoms of fatigue and lethargy. The doctor suspects that, for some reason, blood sugar levels are abnormally low. But testing indicates that blood sugar levels are normal. Further testing reveals that sugar levels inside cells are indeed abnormally low. a. What could be going on here? Why might the sugar levels inside the cells be low even though in the bloodstream they are normal? State in terms of genes. b. One way to explain the situation in part a is that a gene at one locus is affecting the expression of a gene at a…