Which one is correct about somatic mutations? They can be inherited to progeny They can cause cancer They happen during fertilization They cannot be repaired
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HELP PLEASE NO EXPLANATION NEEDED
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Which one is correct about somatic mutations?
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They can be inherited to progeny
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They can cause cancer
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They happen during fertilization
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They cannot be repaired
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Step by step
Solved in 2 steps
- Hi can I get help with this question please? I am stuck!! Topic is sexual reproduction and it’s physiology. Scenario: Hefty Harry is tall, muscular, produces sperm, and he has a thick beard, high concentrations of circulating testosterone, and a penis. In other words, he very much looks like a man. However, when he got his genome sequenced, he and everyone else were extremely surprised to learn that he has two X chromosomes. He comes to ask you how this could be possible. You tell Hefty Harry that it’s possible...(see image)PCOS (polycystic ovarian syndrome ) what's on normal gene function.A couple has had a child born with neurofibromatosis. They come to your genetic counseling office for help. After taking an extensive family history, you determine that there is no history of this disease on either side of the family. The couple wants to have another child and wants to be advised about the risks of that child having neurofibromatosis. What advice do you give them?
- You are in charge of a new gene therapy clinic. Two cases have been referred to you for review and possible therapy. Case 1. A mutation in the promoter of a proto-oncogene causes the gene to make too much of its normal product, a receptor protein that promotes cell division. The uncontrolled cell division has caused cancer. Case 2. A mutation in an exon of a tumor-suppressor gene makes this gene nonfunctional. The product of this gene normally suppresses cell division. The mutant gene cannot suppress cell division, and this has led to cancer. What treatment options can you suggest for each case?Jan is concerned about using ART. She wants to be the genetic mother and have Darryl be the genetic father of any children they have. What methods of ART would you recommend to this couple? Jan, a 32-year-old woman, and her husband, Darryl, have been married for 7 years. They have attempted to have a baby on several occasions. Five years ago, they had a first-trimester miscarriage, followed by an ectopic pregnancy later the same year. Jan continued to see her OB/GYN physician for infertility problems but was very dissatisfied with the response. After four miscarriages, she went to see a fertility specialist, who diagnosed her with severe endometriosis and polycystic ovarian disease (detected by hormone studies). The infertility physician explained that these two conditions were hampering her ability to become pregnant and thus making her infertile. She referred Jan to a genetic counselor. At the appointment, the counselor explained to Jan that one form of endometriosis (MIM 131200) can be a genetic disorder, and that polycystic ovarian disease can also be a genetic disorder (MIM 184700) and is one of the most common reproductive disorders among women. The counselor recommended that a detailed family history of both Jan and Darryl would help establish whether Jans problems have a genetic component and whether any of her potential daughters would be at risk for one or both of these disorders. In the meantime, Jan is taking hormones, and she and Darryl are considering alternative modes of reproduction. Using the information in Figure 16.4, explain the reproductive options that are open to Jan and Darryl.females infertility the germ line less one too many a normal amount of autosomes one too few death males sex chromosomes somatic cells Humans who have aneuploidy of the sex chromosomes are viable. A human who has Turner syndrome (XO) has the normal number of autosomes and will produce the normal amount of proteins from gene on autosomes. A person who is XO has one too few X chromosomes. A Barr body will not form. Most X chromosome genes produce a normal amount of protein. The few genes in the PAR region of the X chromosome remain active on all copies of the X chromosome. In Turner syndrome, those genes produce somatic cells than normal, leading to skeletal abnormalities. with protein in In the Barr body is normally reactivated. In individuals with Turner syndrome, the lower dose of X chromosome gene expression in the female germ line leads to defective oogenesis and
- Haploinsufficiency means the reduced dosage of a normal gene product is not enough for a normal phenotype TRUE OR FASLE i need explanationLet us practice it again! Analyze the pedigree below to answer the questions that follow. Huntington's disease a disorder in which nerve cells waste away, or disintegrate, is passed down through families. certain parts of the brain Huntington's diseate llustration ereated in htps://pregenygenetion.com/ 1. What members of the family above are affected with the Huntington's disease? 2. Tnere are no carriers ior Huntungton's disease you either have it or you do not. Is Huntington's disease caused-by a dominant or recessive trait? 3. Identify the genotypes of the following individuals using the pedigree above. (homozygous dominant, homozygous recessive, heterozygous). I- 1 II -1: II -3: III - 4 : 4. How many children did individuals I-1 and I-2 have? 5. How many girls did II-1 and II-2 have? How many have Huntington's Disease? 6. How are individuals III-2 and II-4 related? I-2 and III-5?I need this to be : to be shown as superscript on the X chromosomes
- E Samu E Samu FAIPQLSCDH_No2]MEaHUdrt5NWa2uY6AaKa7Bhy4xfhwplF-CsVAbyA/formResponse Gmail YouTube O Maps Which model below accurately depicts the process of mitosis? * 2n 2n 2n Option 1 Option 2 2n 2n 2n 2n Option 4 Option 3DRAW OUT CLEARLY each of these chromosomes labeling as appropriate (demoed in class) The cell has a diploid number of 8 Each pair has a set of linked genes that are located on each end of the chromosome (paternal on left, maternal on right) Pair 1 AB and ab Pair 2 DE and de Pair 3 Fg and fG Pair 4 hI and Hi Show what the chromosomes would look like in G1, G2, TI and TIIsomatic cells males a normal amount of one too many sex chromosomes excess females infertility death the germ line autosomes one too few Humans who have aneuploidy of the sex chromosomes are viable. A human who has the Klinefelter syndrome (XXY) has the normal number of and will produce the normal amount of proteins from gene on autosomes. A person who has Klinefelter syndrome (XXY) has X chromosomes, one of which will condense into a Barr body in somatic cells. Most X chromosome genes produce protein. The few genes in the PAR region of the X chromosome remain active and those genes produce XXY somatic cells, leading to skeletal abnormalities in with Klinefelter syndrome. protein in the In the Barr body is reactivated. The higher dose of X chromosome gene expression in the XXY male germ line leads to defective spermatogenesis and
