Hyperacetylation of histone tails is often associated with: Question 22 options: Tight conformation of chromatin Loose conformation of chromatin
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Question 22 options:
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Tight conformation of chromatin
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Loose conformation of chromatin
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- "The four core histones are relatively small proteins with a very high proportion of positively charged amino acids; the positive charge helps the histones bind tightly to DNA, regardless of its nucleotide sequence" is true or false.Heterochromatin and Euchromatin Have Which Different Histone modificationsDescribe the packaging of chromosomal DNA by histones with diagrammatic representations. Name the various histone modifications and describe any two among them.
- The activity of ________ will result in an increasedpositive charge on the histone. Question 24 options: Histone acetyltransferase Histone deacetylase Histone methyltransferase Maintenance methylase Histone demethylaseDescribe the basic structure of a nucleosome. What is the role of histone H1?Discuss the levels of chromosomal organization with reference to the following terms: a. nucleotide b. DNA double helix c. histones d. nucleosomes e. chromatin
- Spliceosomes include all of the following EXCEPT enzymes catalyzing acetylation of histone proteins snRNPs RNA-annealing proteins ATP-dependent RNA-unwinding proteinsWhich statement is FALSE? Group of answer choices Histones are very conserved at the primary sequence level in eukaryotes. Histones H2A, H2B, and H1 all contain the histone fold domain (HFD). CENPA is most closely related to histone H3. The amino terminal tail of histone H3 is the location of most of its covalent modifications. Amino terminal ends of core histones are involved in internucleosome interactions.Which histone is not a necessary component in the formation of nucleosornes?
- Eukaryotic Genetic Sequence: 5'-TAC CAT GAT CCC TAT - 3' 1. What would be the newly synthesized DNA strand and explain how the strand will be replicated. Where in the cell would this occur? 2. What would be the synthesized mRNA strand, and how is it transcribed from the original DNA strand, and then converted from a pre-mRNA strand to a mature mRNA? Where in the cell does this occur? 3. What would be the anti-codons for the tRNA. What are the amino acids generated based on the RNA. How are these amino acids translated into protein and where in the cell does this happen?What does histone acetylation favor?The tumor suppressor pRB also binds to and suppresses the activity of retinoblastoma-binding protein 2 (RBP2), a histone demethylase that removes methyl groups from di- and trimethylated lysines in histone 3. What is the possible consequence of an inactivating mutation in RB1 that causes an inability of pRB to bind RBP2?